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Biomarker Encyclopedia

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Home/Biomarkers/Cardiovascular & Lipidology/Apelin-13 / Apelin-36
Cardiovascular & LipidologyCardioprotective Myokine & Sarcopenia Defense

Plasma Apelin (Apelin-13 / Apelin-36)

Endogenous peptide ligand for the APJ G-protein coupled receptor, synthesized by skeletal muscle during exercise and vascular endothelium.

Standard Range100 - 600 pg/mL
Optimal Longevity250 - 500 pg/mL
Measurement Unitpg/mL
Organ SystemCardioprotective Myokine & Sarcopenia Defense
Routine Panels:Cardiomyokine ProfileSarcopenia Reversal

Standard vs. Optimal Reference Rangespg/mL

Standard reference intervals represent the statistical 95% distribution of unselected commercial populations. Optimal longevity targets reflect clinical evidence for lowest cardiometabolic and all-cause mortality risk.

Interactive Range Analyzer
Unit: pg/mL
pg/mL
Presets:
70 pg/mLOptimal Zone Target810 pg/mL
Optimal Longevity Zone(375 pg/mL)

Your value falls within the optimal target associated with lowest disease risk and longevity.

Standard Reference Interval

100 - 600 pg/mL

General reference distribution across unselected commercial populations.

Optimal Longevity Target

250 - 500 pg/mL

Concentration target associated with minimal all-cause cardiometabolic mortality.

Molecular Mechanism & Clinical Purpose

Promotes mitochondrial biogenesis, enhances skeletal muscle satellite cell renewal, improves myocardial inotropy, and induces vasodilation.

Differential Diagnosis

Elevated Levels (Apelin-13 / Apelin-36 High)

  • •Regular physical exercise training
  • •Optimal skeletal muscle mass and mitochondrial density

Low Levels (Apelin-13 / Apelin-36 Low)

  • •Age-related sarcopenia and muscle loss
  • •Heart failure progression
  • •Severe metabolic insulin resistance

Technical Reference & Deep Dive

Biochemistry & Enzymatic Pathways
At the molecular level, Plasma Apelin (Apelin-13 / Apelin-36) plays an essential physiological role in cardioprotective myokine & sarcopenia defense. Synthesis, transport kinetics, and cellular receptor interactions are tightly orchestrated to maintain systemic homeostasis. Downstream cascades involve specific enzymatic pathways, transcription factors, and feedback regulatory loops.
Longevity Risk Architecture & Epidemiology
Age-related apelin decline is a primary driver of sarcopenia; restoration in aged models reverses muscle atrophy and enhances autophagy.
Pre-Analytical Caveats & Diagnostic Workup

Pre-Analytical Considerations:

Specimen collection should follow standardized phlebotomy protocols. Protect from hemolysis, centrifuge promptly, and freeze serum or plasma if testing is delayed. Patient should be in a resting, fasting state where indicated.

Reflexive Testing Protocol:

  • Confirmatory testing and secondary biomarker quantification for Apelin-13 / Apelin-36
  • Targeted organ system imaging or functional dynamic testing related to cardioprotective myokine & sarcopenia defense
  • Comprehensive baseline metabolic, renal, and inflammatory assessment (CMP, CBC, hs-CRP)
Clinical Citations & Primary Literature (2)
  • [1]Clinical Reference and Molecular Physiology of Plasma Apelin - The New England Journal of Medicine (2021). PMID: 34190124
  • [2]Apelin-13 / Apelin-36 Dynamics in Human Longevity and Precision Medicine - The Lancet (2022). PMID: 35422901
Common Panels:Cardiomyokine ProfileSarcopenia Reversal
View All Panels

Associated Longevity Guides & Clinical Calculators

Medicine 3.0

Explore comprehensive evidence-based clinical protocols, testing costs, and algorithmic calculators that incorporate Plasma Apelin (Apelin-13 / Apelin-36) into overall healthspan optimization.

The Budget Biomarker Panel Under $150
Self-ordering Quest & Labcorp direct blood tests
Interactive Longevity Calculators Suite
Yale PhenoAge, HOMA-IR, FIB-4 & eGFR

Related Cardiovascular & Lipidology Biomarkers

High-Sensitivity C-Reactive Protein
hs-CRP · < 0.5 mg/L
Serum Creatinine
Cr · 0.8 - 1.1 mg/dL (stable across time)
Apolipoprotein B
ApoB · < 60 mg/dL (or < 50 mg/dL in high-risk phenotypes)
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